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especially among children aged 1-3 [years], was created when [vaccine] coverage was low, and the apparent interruption of wild poliovirus circulation limited the acquisition of natural immunity [in the well-vaccinated southern health region]." The same situation of poliomyelitis occurring in fully vaccinated children, usually straight after mass vaccination drives, has occurred in many other countries, both developed and developing. The difference was in the truthfulness in reporting. especially among children aged 1-3 [years], was created may multiply and produce new infectious virus. They when [vaccine] coverage was low, and the apparent have therefore discarded the term ‘killed’ and adopted interruption of wild poliovirus circulation limited the the word ‘inactivated’ to replace it. Even ‘inactivated’, acquisition of natural immunity [in the well-vaccinated however, is used in a restricted sense; it refers to the southern health region|]." loss of viral infectivity—that is, to the inability of the The same situation of poliomyelitis occurring in fully virus particles to multiply and produce a new infectious vaccinated children, usually straight after mass _ virus in susceptible cells, when these cells each receive vaccination drives, has occurred in many other _ only single particles of the inactivated preparation, and countries, both developed and developing. The no other virus particles or derivates thereof." More difference was in the truthfulness in reporting. recently, inactivation has been used as a method of studying the structure and function of viruses. "This Mechanics of vaccine-associated paralysis approach received its principal stimulus from the There is more than one aspect to the mechanics of _ discovery that inactivation was sometimes reversible." vaccine-associated paralysis. One of the most (Fenner, 1962) important suspects is increased neurovirulence Little attention has been given to viral intracellular associated with a single nucleotide reactivation. Multiplicity of change in a noncoding type 3 reactivation of UV-irradiated influenza poliovaccine genome. virus was demonstrated in 1951, and Evans et al. (1985) wrote: "Most o cross-reactivation was shown to occur he small number of cases o with the same virus in 1956 and 1961. poliomyelitis which occur in countries Not only has mass Recently-irradiated vaccinia virus was where Sabin's attenuated poliovirus polio vaccination shown to undergo bot n multiplicity vaccines are used are temporally and cross-reactivation (Fenner, 1962). associated with administration o not eradicated (In my opinion, this shows the fallacy vaccine and involve polioviruses o aral tic of irradiating food: irradiated bacteria ypes 2 and 3. Recent studies have . Pp Mi . are only temporarily weakened and provided convincing evidence that the poliomyelitis, It has revert to their original virulence.) In Sabin 2 and 3 viruses themselves may 1936, Berry and Dedrick (quoted by revert to a neurovirulent phenotype caused a number of Fenner, 1962) had already on passage in man [meaning outbreaks of demonstrated that some rabbits babies] ... a point mutation in 9 Q inoculated with a mixture of heat- he 5' noncoding region of the paralysis directly inactivated myxoma virus and genome of the poliovirus type 3 linked to the active fibroma virus died o vaccine consistently reverts to 99 myxomatosis. These data wild type in strains isolated from administered emphasise the dangers o injecting dubiously "inactivated" or "non-genetically" reactivated viruses. Published orthodox medica literature has documented many outbreaks of paralysis connected demonstrable increase in the with mass vaccination programs neurovirulence of the virus." time and again and in many Inherent problems with inactivation of viruses countries. In this article, I've only described a few (including those contaminating polio vaccines) were examples, but it would be easy for the interested reader already known as early as 1961 and 1962. to search the existing literature for more examples. Gerber et al. (1961) described inactivation with formaldehyde which is subject to asymptotic factor, | Conclusions cases of vaccine-associated poliomyelitis. Virus with this change is rapidly selected on passage through the human gastrointestinal tract. The change is associated with a vaccines. meaning that within about 40 hours most viruses are Not only has mass polio vaccination not eradicated inactivated but afterwards there is a viable residue of _ paralytic poliomyelitis, it has caused a number of live viruses indefinitely. outbreaks of paralysis directly linked to the Fenner (1962) described reactivation of animal administered vaccines. viruses: "It is still a common practice among medical These days, when a vaccinee develops poliomyelitis, it men [and women] to speak of ‘killed’ and ‘live’ viral may not be called poliomyelitis; instead, it may be vaccines, and the everyday meaning of the terms is clear called viral or aseptic meningitis, ascending paralysis enough. But, as I shall demonstrate, virologists now (Guillain-Barré syndrome), cerebral palsy (over 75 per recognise a variety of situations in which ‘killed’ virus cent of cases are not diagnosed at birth but after six paralytic poliomyelitis, it has caused a number of paralysis directly linked to the vaccines. Conclusions Not only has mass polio vaccination not eradicated paralytic poliomyelitis, it has caused a number of outbreaks of paralysis directly linked to the administered vaccines. These days, when a vaccinee develops poliomyelitis, it may not be called poliomyelitis; instead, it may be called viral or aseptic meningitis, ascending paralysis (Guillain-Barré syndrome), cerebral palsy (over 75 per cent of cases are not diagnosed at birth but after six 30 * NEXUS Not only has mass polio vaccination " not eradicated outbreaks of administered OCTOBER - NOVEMBER 2009 www.nexusmagazine.com