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EFAs, Oxygenation and Cancer Prevention Continued from page 24 An article in Human Nutrition: Clinical About the Author: Nutrition (1984; 38C:245-260) further Brian Scott Peskin, BSEE, earned his So, it is not cholesterol itself that clogs verifies that it is parent omega-6 that Bachelor of Science degree in Electrical the arteries. If you have a deficiency of | makes up most of the fatty acids in LDL Engineering from Massachusetts Institute EFAs, cholesterol acts as a "poison and HDL cholesterol.” of Technology (MIT) in 1979. He delivery system". EFAs are cholesterol's Don't let anyone ever tell you that founded the field of Life-Systems major component. As the medical natural fats are "bad". One hundred Engineering Science in 1995. Brian was textbook Molecular Biology of the Cell trillion cells need lots of EFA-containing appointed as an adjunct professor at makes clear (p. 481), cholesterol is patural fats; in particular, lots of parent Texas Southern University in the necessary for the structural integrity of the omega-6. Department of Pharmacy and Health lipid bi-layer, the matrix in each of our Sciences in 1998-1999. Brian 100 trillion cell membranes. JAMA (1994; eventually started his own company, 272:1335-40) published an article stating Maximum Efficiency Products, so he that cholesterol-lowering drugs do not could publish his scientific findings and work significantly to prevent heart promote his unique nutritional If just a little of this parent omega-6 is defective, reducing its ability to absorb oxygen and perform other cellular functions, it acts as a direct cause of . cancer as well as heart disease. oo . disease. The reason? They can't lower seen aS RS EE OSASE supplements. Today he is an the amount of defective parent omega-6 Author's Note: independent researcher. ; a enough. on nninl than, This article is based on information in Special thanks to Kenneth Sperling, Khanada Taylor and Jill Kostrinsky for their editorial assistance. As stated in Current Atherosclerosis The Hidden Story of Cancer, written by Reports (2004; 6:477-84), this is why Brian Peskin with clinical researcher cholesterol drugs can't do the job:” "LDL Amid Habib, MD, FAAP, FACE (Pinnacle contains up to 80% lipids [fats and oils], Editor's Note: Press, 2006, see review in NEXUS including polyunsaturated fatty acids and Dye to space constraints, we cannot 13/04). The book is available from cholesterol, mainly esters. Linoleic acid publish the full text of Prof. Peskin's Pinnacle Press, PO Box 56507, (LA), one of the most abundant fatty acids article together with tables and endnotes Houston, TX 77256, USA, or by phoning in LDL..." (consequently, there are gaps in the 1800 456 9941 (toll-free in North With this information, we see that itis endnote numbering). The complete America) or +1 (713) 979 0065. For what the cholesterol is transporting—the article can be viewed at more information, visit the website adulterated EFAs—that is the problem. _ http://www.BrianPeskin/Nexus.com. http:/Avww.BrianPeskin.com. An article in Human Nutrition: Clinical Nutrition (1984; 38C:245-260) further verifies that it is parent omega-6 that makes up most of the fatty acids in LDL and HDL cholesterol.” Don't let anyone ever tell you that natural fats are "bad". One hundred trillion cells need lots of EFA-containing natural fats; in particular, lots of parent omega-6. If just a little of this parent omega-6 is defective, reducing its ability to absorb oxygen and perform other cellular functions, it acts as a direct cause of cancer as well as heart disease. oo So, it is not cholesterol itself that clogs the arteries. If you have a deficiency of EFAs, cholesterol acts as a "poison delivery system". EFAs are cholesterol's major component. As the medical textbook Molecular Biology of the Cell makes clear (p. 481), cholesterol is necessary for the structural integrity of the lipid bi-layer, the matrix in each of our 100 trillion cell membranes. JAMA (1994; 272:1335-40) published an article stating that cholesterol-lowering drugs do not work significantly to prevent heart disease. The reason? They can't lower the amount of defective parent omega-6 enough. As stated in Current Atherosclerosis Reports (2004; 6:477-84), this is why cholesterol drugs can't do the job:* "LDL contains up to 80% lipids [fats and oils], including polyunsaturated fatty acids and cholesterol, mainly esters. Linoleic acid (LA), one of the most abundant fatty acids in LDL..." With this information, we see that it is what the cholesterol is transporting—the adulterated EFAs—that is the problem. Author's Note: Special thanks to Kenneth Sperling, Khanada Taylor and Jill Kostrinsky for their editorial assistance. Editor's Note: Due to space constraints, we cannot publish the full text of Prof. Peskin's article together with tables and endnotes (consequently, there are gaps in the endnote numbering). The complete article can be viewed at http://www. BrianPeskin/Nexus.com. NEXUS + 77 DECEMBER 2006 — JANUARY 2007 www.nexusmagazine.com