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Since sugar consumption per capita in the United States has impacts of high adrenochrome conversion from adrenaline are the increased by roughly a factor of 20 since 1822,” hypoglycaemia numerous interactions that normally occur between triiodothyro- has become rampant in its population.“ As mentioned above, nine, serotonin and the three major components of the antioxidant many recovered schizophrenics feel they had previously suffered defence system. In chronic schizophrenics who have suffered for from hypoglycaemia. Schauss” estimated that between 80 and 85 years, these biochemical abnormalities result in brain atrophy, per cent of criminals in US prisons suffer from hypoglycaemia, associated with large fluid-filled spaces known as ventricles, and often eating an excess of sugary foods and repeatedly drinking serious damage to the thyroid gland. sugar-sweetened coffee and/or Kool-Aid. It is well known that when blood sugar levels drop, adrenaline © TREATMENT AND PREVENTION: EIGHT STEPS is released from the adrenal glands because it is involved in the If the adrenochrome hypothesis is correct, the "ideal" treatment metabolism of glucose.**' It follows, therefore, that anyone for schizophrenia should involve eight steps, designed to reduce suffering from the large blood-sugar swings characteristic of the production of adrenaline and slow down its metabolism to hypoglycaemia (associated with a diet that is too rich in sugar) is adrenochrome and other toxic indoles. Such a treatment should going to overproduce adrenaline. also attempt to reduce the further bio- Hypoglycaemic individuals with one or chemical abnormalities that result more of the genetic aberrations seen in from either an excess. of schizophrenia are therefore likely to A A A adrenochrome and its metabolites or suffer psychosis caused by Since many schizophrenics other impacts of the four genetic aber- adrenochrome created by the oxidation are overoxidising adrenaline rations that appear associated with of this excess adrenaline. due to allergic reactions this mental illness. 3 Since many schizophrenics are BIOCHEMICAL AND CLINICAL they need extra-special overoxidising adrenaline due to aller- IMPACTS OF ADRENOCHROME . gic reactions, they need extra-special Besides being an hallucinogen, surroundings because of surroundings because of such sensi- adrenochrome is a highly reactive neu- such sensitivities. tivities. Ideally, a treatment clinic rotoxin that, in schizophrenia, under- mines at least three major biochemical systems.” It is an antagonist of the hor- mone triiodothyronine and can and often would be like the Lange Meridian Centre, which was built using Bau- biologie principles.** Step two involves genetic and biochemical does seriously damage the thyroid. In chronic schizophrenics, screening to identify the most likely effective treatment protocol. this gland impairment appears to be permanent. Adrenochrome Allergy testing is also essential, as is a low-sugar diet.“ The fifth also has a Jekyll and Hyde relationship with serotonin and so step in the treatment of schizophrenia should involve medications impacts on tryptophan and its other chief metabolite, niacin. At that must quickly reduce the destructive impacts of excess low levels, serotonin appears to stimulate adrenochrome forma- adrenochrome and its derivatives. They must also address the tion, while at higher levels it retards the process. other biochemical anomalies directly related not to such indoles, Adrenochrome also generates numerous free radicals, causing but to the genetic aberration encouraging their overproduction. oxidative stress, eventually exhausting the schizophrenic antioxi- In schizophrenics with the MTHFR C677TT variant, for example, dant defence systems, creating deficiencies of glutathione peroxi- the patient will also be suffering from depressed methionine and dase, superoxide dismutase and catalase. Complicating the elevated homocysteine levels. Endnotes analysis of cerebrospinal fluid biogenic Research Communications 281:267-271 1. John Paul II, Letter to the President of amines in schizophrenia: I. Comparisons 12. Deng, H., Liu, X., Cai, G., Sun, X., the Second World Assembly on Ageing with healthy control subjects and Wang, Y., Terwedow, H., Wang, Z. and Xu, (Madrid, 8-12 April 2002), http://www. neuroleptic-treated/unmedicated pairs X. (2002), "A linkage disequilibrium study vatican.va/holy_father/john_paul_ii/letters/ analysis", Psychiatry Research of methylenetetrahydrofolate reductase 2002/documents/hf_jp-ii_let_20020410_ 52(3):237-249 C677T and schizophrenia", Zhonghua Yi assembly-ageing_en.html 7. Myers, D.G. (1992), Psychology, Worth Xue Yi Chuan Xue Za Zhi 19(3):198-200 2. Torrey, E.F. (1980), Schizophrenia and Publishers, New York 13. Novak, G., Kim, D., Seeman, P. and Civilisation, Jason Aronson, New York 8. ibid. Tallerico, T. (2002), "Schizophrenia and 3. Fishbein, M. (1985), Fishbein's 9. Nicol, S.E. and Gottesman, I.I. (1983), Nogo: Elevated mRNA in cortex, and high Illustrated Medical and Health "Clues to the genetics and neurobiology of prevalence of a homozygous CAA insert", Encyclopaedia, Stuttman, Westport, CT schizophrenia", American Scientist 71:398- Molecular Brain Research 107(2):183-189 4. Horrobin, D. (2002), The Madness of 404 14. Foster, H.D. (2003), What Really Adam and Eve: How Schizophrenia Shaped 10. Park, T.W., Yoon, K.S., Kim, J.H., Causes Schizophrenia, Trafford Publishing, Humanity, Transworld Publishers, London Park, W.Y., Hirvonen, A. and Kang, D. Victoria, BC 5. Seeman, P., Guan, H.C. and Van Tol, (2002), "Functional catechol-O-methyl- 15. Hoffer, A. and Foster, H.D. (2000), HLH. (1995), "Schizophrenia: Elevation of transferase gene polymorphism a suscepti- "Why schizophrenics smoke but have a low dopamine D4-like sites, using [3H] nemon- _ bility to schizophrenics", European Neuro- _ incidence of lung cancer: Implications for apride and [1251] epidepride", European psychopharmacology 12(4):299-303 the treatment of both disorders", Journal of Journal of Pharmacology 286(2):R3-5 11. Harada, S., Tachikawa, H. and Orthomolecular Medicine 15(3):141-144 6. Issa, F., Gerhardt, G.A., Bartko, J.J., Kawanishi, Y. (2001), "Glutathione S-trans- 16. Foster, H.D. and Hoffer, A., Sudath, R.L., Lynch, M., Gumache, P.H., ferase M1 gene deletion may be associated "Schizophrenia and cancer: The Freedman, R., Wyatt, R.J. and Kirch, D.G. with susceptibility to certain forms of schiz- adrenochrome-balanced morphism", (1994), "A multidimensional approach to ophrenia", Biochemical and Biophysical Medical Hypotheses (in press) impacts of high adrenochrome conversion from adrenaline are the numerous interactions that normally occur between triiodothyro- nine, serotonin and the three major components of the antioxidant defence system. In chronic schizophrenics who have suffered for years, these biochemical abnormalities result in brain atrophy, associated with large fluid-filled spaces known as ventricles, and serious damage to the thyroid gland. Since many schizophrenics are overoxidising adrenaline due to allergic reactions, they need extra-special wi... is surroundings because of such sensitivities. 30 + NEXUS APRIL — MAY 2004 www.nexusmagazine.com