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be poured along the head/backline of the cow, just millimetres I was also fascinated to discover that these TSE-affected popu- from where the prion protein is manufactured in the cell lines of _lations lived in areas that were enduring front-line exposure to the spinal cord. intensive shock-bursts of low-frequency infrasound—from mili- It was well recognised that OP insecticides exert their toxic tary and quarry explosions, volcanic and earthquake activity, low- effects in mammals by deforming the molecular shape of various flying supersonic jets, Concorde overflights, etc. nerve proteins like acetylcholinesterase, whereupon these mal- A specific environmental source of manganese could be pin- formed proteins cease to perform their proper function in the pointed in every TSE cluster zone that I had investigated to date, brain. But nobody had ever considered that a similar-style molec- where each TSE-affected ecosystem could be directly connected ular interaction might occur between OPs and the prion protein. to the atmospheric fallout of some naturally occurring or industri- Since the prion protein has been shown to bond up with copper in al source of combusted manganese oxide, e.g., stemming from the healthy brain, I felt that the ability of these dithio insecticides volcanic activity, acid rain, steel/glass/ceramic/dye/munitions fac- to lock up copper in the treated animal may play some role in tories, lead-free petrol refineries, the take-off airspace beyond air- deforming the prion protein. ports, etc. In this respect, it should be borne in mind that atmos- After many abortive attempts to coerce the Establishment into —_ pheric manganese, much like silver and aluminium, can be running the correct laboratory test, I eventually raised funds from absorbed directly into the brain via the nasal-olfactory inhalatory well-wishers and obtained personal loans to finance Dr Stephen route of intake. Perhaps this highly efficient mode of uptake Whatley of the Institute of Psychiatry in enables sufficient concentrations of man- London to challenge brain cell cultures with ganese to accumulate in the brain and initiate the OP phosmet—the actual OP used at TSE? uniquely high doses on UK farms. Furthermore, many of the mammalian Amazingly, these trials demonstrated that populations involved in the outbreaks of both the OP altered the cellular metabolism of traditional and new-strain TSE could be prion protein in some of the ways observed linked to the consumption of high- in the early stages of spongiform disease— concentration manganese supplements for suggesting that phosmet exposure may ren- A specific bone growth, etc. For instance, the clusters der mammals more susceptible to the dis- . of chronic wasting disease in deer could all ease. Unfortunately, these experiments did environmental be linked to the areas where a "spiced up" not produce the key deformation of the prion source of manganese manganese mineral lick had been put down protein that is seen in TSEs. . . by the deer hunters for addicting deer to their I returned to square one, assuming that could be pinpointed shooting territories. The manganese is added OPs in combinati ith a further factor X i f ting antl th. In fact, all could fulfill the final missing link in in every TSE ms species connected with TSEs—deer, the causal jigsaw. Or perhaps OPs cluster zone that cows, goats, mink, sheep, zoo animals, weren't involved at all! I h . 7 cats, humans, etc.—are_ given ad investigated thor f manganese supplements in their feed. The Cluster Buster to date. Disturbingly, manganese is also Caught in the midst of a minefield of multinational interests, medical spin doctors and political propagandists, I grew exhausted by the vortex of pro- fessionals who had successfully hijacked all UK scientific research into TSEs into a cul-de-sac. Furthermore, I'd found myself frozen added to milk substitute powders for calves (and human infants) at levels up to 1,000 times those found in normal cow's milk. This practice has been widespread in all countries affected by BSE to date. Excessive intake of dietary man- ganese poses a great risk for the imma- out into the cold, operating much like ture mammal, since the regulatory an underground scientist, tramping a mechanisms of the blood-brain barrier covert journey around the rustic outbacks of the UK's farmlands are underdeveloped at this stage, thereby permitting an excessive to nail down the true cause of BSE. uptake of manganese and other metals into the brain. So I expanded my horizons and embarked upon a refreshing The addition of manganese to substitute milk powders explains ecodetective trek to analyse the unique environments around the why European dairy cattke—which were invariably reared on this world where traditional TSEs had erupted as high-incidence clus- powder—suffered such high incidence rates of BSE in relation to ters for many years. By scanning the overall essence of each clus- the negligible rates in beef suckler and organically reared cattle— ter location, I attempted to pinpoint the common causal factors— which were invariably reared on natural cow's milk. the aetiological needles in the causal haystack. Against a backdrop of flamboyant and sometimes threatening Every Storm Cloud Has A Silver Lining scientific scenery, I embarked on a lone journey to the ends of the A few other TSE cluster hotspots had demonstrated the same Earth, sampling exotic corners of Colorado, Iceland, Slovakia, low-copper connection but had measured abnormally high levels Calabria, Sardinia, Japan, etc.—areas where an assortment of ani- of other potentially toxic transition metals such as silver, platinum mals and humans had demonstrated a high incidence rate of TSE. and lithium as well as manganese. Much like manganese, these My analytical results displayed abnormally high levels of the metals will also readily substitute at copper bonds on prion metal manganese and rock-bottom levels of copper, selenium and _ proteins. zinc in common in all of these food chains. Levels of manganese Some of these TSE environments were silver-mining areas were normal in adjoining disease-free areas. where local ecosystems were naturally high in silver, while others A specific environmental source of manganese could be pinpointed in every TSE aan ee ee | had investigated to date. Every Storm Cloud Has A Silver Lining A few other TSE cluster hotspots had demonstrated the same low-copper connection but had measured abnormally high levels of other potentially toxic transition metals such as silver, platinum and lithium as well as manganese. Much like manganese, these metals will also readily substitute at copper bonds on prion proteins. Some of these TSE environments were silver-mining areas where local ecosystems were naturally high in silver, while others 28 = NEXUS APRIL — MAY 2003 cluster zone that www.nexusmagazine.com