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DENTAL AMALGAM & MERCURY POISONING 24. Ibid. 25. Selinger, B., Chemistry in the Market Place, Canberra, Aust. National University Press, 1978, p387. 26, Buist, R., Food Chemical Sensitivity, Sydney, Harper and Row Publishers, 1986, p.206. See also Cox and Eley, p.262. 217. Anon., Choice, August 1983, p28. 28, Farrer, K.TH., additives and contaminants, Carlton, 7 ‘Melbourne. University Press, 1983, p.40. 29. Davey, H.A. and Van Moort, J.C., "Current Mercury Deposition at Ngawha Springs, New Zealand”, Search, Vol.5 No.4, 1974, p.156. 30. Hichom, J. "Dental Amalgam and Mercary Toxicity", No.18, 1987, p.93. 31. Mylander, M., Friberg, L. and Lind, B., "Mercury Concentrations in the Human Brain and Kidneys in Relation to exposure from Dental Amalgam Fillings", Swed, Dent. J., Val.11, 1987, p.180. 32. Gay, D.D., Cox, R.D., and Reinhardt, J.W., "Chewing Releases Mercury From Fillings”, The Lancet, May 5, 1979, p.985. 33. Langan et.al., pp.870-871. 34. Tbid., p.869. 35. Eley, B.M. and Cox, S.W., “Mercury Form Dental Amalgam Fillings in Patients”, British Dental Journa}, October 10, 1987, p222. 36. Bid. p221. 37. Ayres, S., "Amalgam and Gold Dental Fil Fillings: Reaction from Electrogalvanic Current", Dermatology, Vol.14, No.2, 1986, p277. See also Ayres, S., “Sore mouth caused by electrogalvanic current”, J, of the Rova) Society of Medicine, Vol.77, August 1984, p.709. 38. Langan etl, p.871. 39. Ayres, 1986. 40. Pleva, J., "Mercury Poisoning and Dental Amalgam", Vol.12, No3, 1983, p.190. 41. Eley and Cox, p.222. 42. Ibid, sce also Eley and Cox. REFERENCES: 1, Dili, PA. and D'Ini, FM., i ‘Tragedy, New York, John Wiley & Sons, 1977, pp.181- 191. 2. Dbid., p.187 3. Ibid. p.186 4, Ibid. p.188. 5. Langan, D.C., Fan, PLL., and Hoos, A.A., “The Use of Mercury in Dentistry: A Critical Review of the Recent Literature", The Journal of the American Dental Association, Vol.115, December 1987, p867. See also D'ftri and D'tti, p.211. 6 Ibid. 7. Tid, 8. Ziff, S., The Toxic Time Bomb, Wellingborough, Northamptonshire, Thorsons Publisters Limited, 1985, p.11. 9. Langan ¢t.al., p.867. 10. Ziff, pp.11-12. TL. Langan etal, p.868. See also Ziff. 12. D'ltn and D'lui, pp.29-46, and 139-168. 13. Fritsch, A.J., The Household Pollutants Guide, New York, Anchor Press, 1978, p.210. 14. D'ti and D'lui, pp.202-210. See also Langan eta., p.869. 15, D'led and D'lui, p.47. 16. Dbid., p.16. 17. Ibid., p22. 18. Jensen, S., and Jernelov, A., “Biological Methylation of Mercury in Aquatic Organisms", Nature, Vol.223, 1969, pp.753- 754, 19. Langan eual., p.869. 20. Sokrenats Teter I~ mat oe ape Geveland, Ohic (CRC Press, 1972. 21. Langangtal., p:875\ 22. Cox, $.W., 3 Eley, BM., “Mercury Release, Distribution and Excretion From Subcutaneously Implanted Conventional and High-Copper Amalgam Powders in the Guines Pig", Arch. Oral Biol, Vol.32, Nod, 1987, p.262. 23. Rawls, RL., "Reproductive Hazards in the Workplace” . Chemical and Engineering News, February 18, 1980, p.36. amalgam dental treatment to be considered, A number of well-controlled studies have established that during the filling process with amalgam, considerable quantities of mercury vapour and mercury amalgam par- ticulates are expelled in the oral cavity and breathing zone. Hand condensation of the fillings resulted in mercury levels up to 320 micrograms/m’ with levels up to 770 micro- grams/m? measured with mechanical con- densation. Dry cutting and polishing proce- dures resulted in very high levels, often exceeding 1000 micrograms/m’." During these processes fine particles of amalgam may become accidently embedded in the soft tissues of the mouth. Investigations have shown that these parti- cles of amalgam undergo progressive degra- dation within phagocytic cells. During this process mercury is released from the cells into the tissues. This mercury has been found to pass from the tissue fluid into the Continued in the next issue of Nexus. JUNE-JULY 1992 Continued from page 65