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FLUORIDE CONTAMINATION FROM BEVERAGE CONSUMPTION FLUORIDE CONTAMINATION FROM puport to reveal the actual mchanism by virtue of which fluoride BEVERAGE CONSUMPTION can distup e DNA molecule and the ctive sites of the molecules of may human enzymes. By far the mos common source of additional fluoride intake When all is said, it is manifestly clear that the time has come fo comes from beverage consumpon. Beverages which conain f}uori- a senous and cmprehensive review of the policy which mandates dated water include reconstituted juices, punches, popsicles, other the compulsory fluoridation of our muniipal water supplies. Sudl a water-based frozen desserts and caronated beYerages. Studies have _ review will no doubt which reliable reseuch investigations can be shown thal soft drink consumption in the US has incr mrkedly over _ integrad with a philosophy of health education to assist their imple- the Last two decades7notonly aunong enage boys m 15-17 years of | mentation. Through education it may be possile to appreciate that age, but among 12 year old children. Statistics show that in Cda soft within nature itself are important patlerns of design for an overall drink consumption increased by 37% rom 1972 D 981. e increase programme of health. In nature, for instance fluorides are typicany in soft drink consumplion coincided wih a decrease in the con- found in deidedly luble forms which are relatively safe. By deliber- sumption of milk, thereby increasing the overall luode inlake. A ately intervening make nature’s if orms of fluoride olul, we lans- nurnber of st-ldies reveal that the dramatic increase in beverage fonn a relatively humless natural substance into a concentrated and consumption, coupled wih fluaridation of municipal walers con- _ highly toxic substance which can then be indiscnminately dispersed stibtes a potential health hazard. Prolonged exposure to fluorides _ throughut the environmen as a poison. The subde constelation of my ctully increase raaher han diminish the incidence of tooth deeay. health clues which nature provides m respect of fluoides is further Enzymtic damge related to enamel mineralisation creates a parotic __ illustrated by the simple bllt slegant mechanisms of breast-feeding. looth far more susceptible of caries than would otherwise e the Bast-fed infants are actually potected from reeiving more than case.” extremely low concenaion of fluoide in breast mill by an inbu}lt Ina major study of adverse effects of fluoride Continued on ae 46 physiological plasma/milk aier against fluoride.’° There is much Yiamouyiannis and Burle reported in 1977 that at least 10,000 peo- about health to learn rom nature, bu to do so we must be moe con- ple in he US die every ycr of fluoride-induced cncer. In the intro- cerned to join with nature in partnership than to stand back from duction to theu work 7 esearch papers ae cited which demonstrate — nahJre o suWue and manipulale it. the mugenic effects associated with fluorides. Thcre is now side Whether the flundaion campaign must be indicted in he light of consensus within the scientilc ommunity that the mutagenic tivity _ the evidenc as one of the major public hoaxes perpetrated this cen- of a sllbstance an be regarded as an important indication of its tury, is a judgement est reserved for the reader. Whstever the judge- potential cancer ca;ing activi. ment, it is incontestable that the prevention of tooth decay is not the bottom-line of he fluoridation debate when the pancea has become the poison. REFERENCES FLUORIDATE 1. N.Y: Rinbold Publird Cp., d EL, 1963, p. I 187. 2. Hotes, L., nvi Pollutiont (N.Y.: Holt, R dWut0, 2nd Ed ,1977, p.64. your water 3. Wlcer, G.S.R., Pluoridtion - Poion on T, Melkne Gla Publihor, 198), p.44. 4. Den, ILT., “Sludia Ml Thrho)d of | Sin aa’ Ckenic nic lusi 5, 11 R. 1934: SO: pp.17129 S. W ,p.llS 6. Sto ., “Pluoridiion, I’ Poial?, 7. Cldwell, G. end Z4n, Pl, iLi n Rod Ecol P, 1974), p.7. 8. Ibid 9. mey, W., Fluaidc in Aw Sytncy: Ic d nger, 198), p.14. 10. Wlka, p.IS9. 11. Ibid 12. Gldll p.8. 13. Bt, B., Cn. J . News. Octob 2, 197y, p.S6. 14. Wldbott, G.L., Fluoridaion I’he Or aWrct: Kn Cado PrG c., 197g. 15. Shemll, D.,g.Ju u 7,1980, p.4. ALCOA ADVERTISEMENT - 1950 16. IA, LR., Chn k Er. . 3 nug 28,198, pp.5. 17. Wa, p.1S6. : > : See ae 18. mey, ride il Au Sine ose pro fe studies over a dcads ago, a YaSt :ientific litera- 19. $14, p.103. ture has continued to acumulate which srongly indicates hat the 99 tha, p.104, practice of fluoridating munici water supplies s a dangerous prac- 31, Canminoc Feod Prd d Nuitial Bo rt Nol Ren:h nal, To Ocalg Nlly in Food tice. In 1983 an Australian dental surgeon, G. Smith, reported a shingt D.C: Non! Aademy i, 1973),pp.72-74. number of studies which suggest at thee is ww ses nsk to the pub- 22. KIlte ,p.308. lic of flunde ovedoe. He argues hat “the crucial argument d not con- 23 . Clals, 1. Ild Hve, J.A., UFh4ride Inke an Bvag CON ‘ Canmlmi t. Orl Ipidmol., cern the fluoride lerel in a community water supply per set but — 198, Sd.16: p.14. rather wheher flua ion increases the nsk that cemin people deelop, 24. M-ml, J., Tlbi, M., nd Sg n, H.D., uld C ria PrC SDG in Cnulir Drinlcin bav even for a short time, levels of fluoride in the blood that can dam- —_ OptiarlFluoriduedWr,” niy Da epidmid., Ig87,VoblS 94. age human cells nd systems.t 25. Ibid, p. 295. In 1985 snother Ausalian scientist, M. Diesendorf drew attention 26. Y ay, J. 4d Bwlc, D., Fluaidwnnd go lae of C-nca Mort Ratt lo ificil |;lurid- to the discovery of a whoe new dimension to the health hazsrds "> tuoido, UL-10, 177, pp.102-123 . : : + > . . : - 27. Sndl, G., “Muarid radvod?”, New Scicntist, S My 1983, p.286. associated with he ingeson of fluorides. Sodium fluoride, for exam- 28, Diadalf, M., nde Ne Ri”, Seuch, Vd.16, NoS, IgU, p.129 ple, had been found to cause unscheduled DNA synhesis and chro- . _— ° 0 ee sae : . : : 2g. Ibid. mosonal aberrations in certaia human cells.2’ Other recent studies 0. Smilh, G., p.87. By far the mos common source of additional fluoride intake comes from beverage consumpon. Beverages which conain f}uori- dated water include reconstituted juices, punches, popsicles, other water-based frozen desserts and caronated be Yerages. Studies have shown thal soft drink consumption in the US has incr mrkedly over the Last two decades7notonly aunong enage boys m 15-17 years of age, but among 12 year old children. Statistics show that in Cda soft drink consumption increased by 37% rom 1972 D 981. e increase in soft drink consumplion coincided wih a decrease in the con- sumption of milk, thereby increasing the overall luode inlake. A nurnber of st-ldies reveal that the dramatic increase in beverage consumption, coupled wih fluaridation of municipal walers con- stibtes a potential health hazard. Prolonged exposure to fluorides my ctully increase raaher han diminish the incidence of tooth deeay. Enzymtic damge related to enamel mineralisation creates a parotic looth far more susceptible of caries than would otherwise e the case.” Ina major study of adverse effects of fluoride Continued on ae 46 Yiamouyiannis and Burlc reported in 1977 that at least 10,000 peo- ple in he US die every ycr of fluoride-induced cncer. In the intro- duction to theu work 7 esearch papers ae cited which demonstrate the mugenic effects associated with fluorides. Thcre is now side consensus within the scientilc ommunity that the mutagenic tivity of a sllbstance an be regarded as an important indication of its potential cancer ca;ing activi. Sine ose pro fe studies over a dcads ago, a YaSt :ientific litera- ture has continued to acumulate which srongly indicates hat the practice of fluoridating munici water supplies s a dangerous prac- tice. In 1983 an Australian dental surgeon, G. Smith, reported a number of studies which suggest at thee is ww ses nsk to the pub- lic of flunde ovedoe. Hc argues hat “the crucial argument d not con- cern the fluoride lerel in a community water supply per set but rather wheher flua ion increases the nsk that cemin people deelop, even for a short time, levels of fluoride in the blood that can dam- age human cells nd systems.t In 1985 snother Ausalian scientist, M. Diesendorf drew attention to the discovery of a whoe new dimension to the health hazsrds associated with he ingeson of fluorides. Sodium fluoride, for exam- ple, had been found to cause unscheduled DNA synhesis and chro- mosonal aberrations in certaia human cells.2’ Other recent studies NEXUS - 16 ALCOA ADVERTISEMENT - 1950 YEAR BOOK + OCTOBER/NOVEMBER 1991